Shift work sleep disorder.
Study Design
- 研究类型
- review
- 干预措施
- Shift work sleep disorder. melatonin, light therapy, prescription medications (modafinil/armodafinil); optimized shift schedule
- 对照组
- Placebo
- 效应方向
- Mixed
- 偏倚风险
- Unclear
Abstract
Shift work sleep disorder (SWSD) is a circadian rhythm sleep-wake disorders affecting individuals who work in nonstandard hours, particularly night shifts. It manifests as difficulty sleeping during the day and staying awake during work hours, leading to health issues. SWSD is not universally experienced by all shift workers, with about 30% affected. Diagnosing SWSD involves monitoring sleep patterns and differentiating it from other disorders such as sleep apnea. Prevention and treatment include collective measures such as optimizing shift schedules and individual strategies such as sleep/circadian hygiene, light therapy, melatonin use, and, if necessary, prescription medications. Despite these interventions, the primary recommendation is to switch to daytime work, although this may not be feasible for all workers.
简要概述
Prevention and treatment of shift work sleep disorder include collective measures such as optimizing shift schedules and individual strategies such as sleep/circadian hygiene, light therapy, melatonin use, and, if necessary, prescription medications.
Used In Evidence Reviews
Similar Papers
Journal of sleep research · 2017
European guideline for the diagnosis and treatment of insomnia.
Chronobiology international · 2012
Circadian typology: a comprehensive review.
Journal of clinical sleep medicine : JCSM : official publication of the American Academy of Sleep Medicine · 2017
Clinical Practice Guideline for the Pharmacologic Treatment of Chronic Insomnia in Adults: An American Academy of Sleep Medicine Clinical Practice Guideline.
Movement disorders : official journal of the Movement Disorder Society · 2011
The Movement Disorder Society Evidence-Based Medicine Review Update: Treatments for the non-motor symptoms of Parkinson's disease.
Cell · 1981
Regulation of terminal differentiation of cultured human keratinocytes by vitamin A.
Journal of neuroendocrinology · 2003