Figure 1

![Figure 1: Movement and muscle function assays in dystrophin-deficient C. elegans (dys-1 mutants) demonstrate baseline locomotor deficits compared to wild-type controls. The data establish the severity of muscular dysfunction in this Duchenne muscular dystrophy model.]()

> Source: Rebecca A Ellwood et al. "Sulfur amino acid supplementation displays therapeutic potential in a C. elegans." *Communications biology*, 2022. PMID: [36385509](https://pubmed.ncbi.nlm.nih.gov/36385509/)

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  <img src="" alt="Movement and muscle function assays in dystrophin-deficient C. elegans (dys-1 mutants) demonstrate baseline locomotor deficits compared to wild-type controls. The data establish the severity of muscular dysfunction in this Duchenne muscular dystrophy model." />
  <figcaption>Figure 1. Movement and muscle function assays in dystrophin-deficient C. elegans (dys-1 mutants) demonstrate baseline locomotor deficits compared to wild-type controls. The data establish the severity of muscular dysfunction in this Duchenne muscular dystrophy model.<br>  Source: Rebecca A Ellwood et al. "Sulfur amino acid supplementation displays therapeutic potential in a C. elegans." <em>Communications biology</em>, 2022. PMID: <a href="https://pubmed.ncbi.nlm.nih.gov/36385509/">36385509</a></figcaption>
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