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Ion channel gating mechanisms potentially affected by phytocannabinoid compounds in epileptic neural tissue are illustrated. Voltage-gated sodium and calcium channels represent key targets for anticonvulsant activity.

Figure 291

Diagram

Source Paper

Receptors and Channels Possibly Mediating the Effects of Phytocannabinoids on Seizures and Epilepsy.

Pharmaceuticals (Basel, Switzerland) (2020)

PMID: 32751761

DOI: 10.3390/ph13080174

Cite This Figure

![Figure 291: Ion channel gating mechanisms potentially affected by phytocannabinoid compounds in epileptic neural tissue are illustrated. Voltage-gated sodium and calcium channels represent key targets for anticonvulsant activity.]()

> Source: Lara Senn et al. "Receptors and Channels Possibly Mediating the Effects of Phytocannabinoids on Se." *Pharmaceuticals (Basel, Switzerland)*, 2020. PMID: [32751761](https://pubmed.ncbi.nlm.nih.gov/32751761/)
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  <img src="" alt="Ion channel gating mechanisms potentially affected by phytocannabinoid compounds in epileptic neural tissue are illustrated. Voltage-gated sodium and calcium channels represent key targets for anticonvulsant activity." />
  <figcaption>Figure 291. Ion channel gating mechanisms potentially affected by phytocannabinoid compounds in epileptic neural tissue are illustrated. Voltage-gated sodium and calcium channels represent key targets for anticonvulsant activity.<br>  Source: Lara Senn et al. "Receptors and Channels Possibly Mediating the Effects of Phytocannabinoids on Se." <em>Pharmaceuticals (Basel, Switzerland)</em>, 2020. PMID: <a href="https://pubmed.ncbi.nlm.nih.gov/32751761/">32751761</a></figcaption>
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