Description
Baseline characterization of lipid accumulation in castration-resistant prostate cancer (CRPC) cell lines. The study establishes that lipid metabolism dysregulation is associated with PCa progression following androgen deprivation therapy.
Figure 1
ChartSource Paper
Melatonin inhibits lipid accumulation to repress prostate cancer progression by mediating the epigenetic modification of CES1.Cite This Figure
![Figure 1: Baseline characterization of lipid accumulation in castration-resistant prostate cancer (CRPC) cell lines. The study establishes that lipid metabolism dysregulation is associated with PCa progression following androgen deprivation therapy.]() > Source: Lijie Zhou et al. "Melatonin inhibits lipid accumulation to repress prostate cancer progression by ." *Clinical and translational medicine*, 2021. PMID: [34185414](https://pubmed.ncbi.nlm.nih.gov/34185414/)
<figure> <img src="" alt="Baseline characterization of lipid accumulation in castration-resistant prostate cancer (CRPC) cell lines. The study establishes that lipid metabolism dysregulation is associated with PCa progression following androgen deprivation therapy." /> <figcaption>Figure 1. Baseline characterization of lipid accumulation in castration-resistant prostate cancer (CRPC) cell lines. The study establishes that lipid metabolism dysregulation is associated with PCa progression following androgen deprivation therapy.<br> Source: Lijie Zhou et al. "Melatonin inhibits lipid accumulation to repress prostate cancer progression by ." <em>Clinical and translational medicine</em>, 2021. PMID: <a href="https://pubmed.ncbi.nlm.nih.gov/34185414/">34185414</a></figcaption> </figure>