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Figure 4

Description

Average plasma glucose concentration under constant routine conditions is reduced following a 5-hour delay in meal times, suggesting meal timing significantly influences glucose metabolism rhythms.

More Figures from This Paper

Hormonal response profiles to standardized meals are compared between normal and delayed meal timing conditions, revealing shifts in metabolic hormone rhythms.

Figure 3

Hormonal response profiles to standardized meals are compared between normal and delayed meal timing conditions, revealing shifts in metabolic hormone rhythms.

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Core body temperature rhythms are displayed across experimental conditions, demonstrating that meal timing shifts selectively affect peripheral but not central circadian markers.

Figure 5

Core body temperature rhythms are displayed across experimental conditions, demonstrating that meal timing shifts selectively affect peripheral but not central circadian markers.

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Plasma glucose concentrations during constant routine conditions show a significant reduction after participants experienced delayed meal times, indicating that meal scheduling influences circadian glucose regulation.

Figure 6

Plasma glucose concentrations during constant routine conditions show a significant reduction after participants experienced delayed meal times, indicating that meal scheduling influences circadian glucose regulation.

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Clock gene expression patterns in adipose tissue are compared between the two meal timing conditions, revealing tissue-specific circadian phase shifts.

Figure 7

Clock gene expression patterns in adipose tissue are compared between the two meal timing conditions, revealing tissue-specific circadian phase shifts.

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Per2 and other clock gene transcript levels measured from peripheral blood samples demonstrate delayed phase alignment consistent with the shifted meal schedule.

Figure 8

Per2 and other clock gene transcript levels measured from peripheral blood samples demonstrate delayed phase alignment consistent with the shifted meal schedule.

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Summary model of how meal timing acts as a zeitgeber for peripheral clocks independent of the central suprachiasmatic nucleus pacemaker.

Figure 9

Summary model of how meal timing acts as a zeitgeber for peripheral clocks independent of the central suprachiasmatic nucleus pacemaker.

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Figure 4

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Source Paper

Meal Timing Regulates the Human Circadian System.

Current biology : CB (2017)

PMID: 28578930

DOI: 10.1016/j.cub.2017.04.059

Cite This Figure

![Figure 4: Average plasma glucose concentration under constant routine conditions is reduced following a 5-hour delay in meal times, suggesting meal timing significantly influences glucose metabolism rhythms.](https://pdfs.citedhealth.com/figures/28578930/246.png)

> Source: Sophie M T Wehrens et al. "Meal Timing Regulates the Human Circadian System.." *Current biology : CB*, 2017. PMID: [28578930](https://pubmed.ncbi.nlm.nih.gov/28578930/)
<figure>
  <img src="https://pdfs.citedhealth.com/figures/28578930/246.png" alt="Average plasma glucose concentration under constant routine conditions is reduced following a 5-hour delay in meal times, suggesting meal timing significantly influences glucose metabolism rhythms." />
  <figcaption>Figure 4. Average plasma glucose concentration under constant routine conditions is reduced following a 5-hour delay in meal times, suggesting meal timing significantly influences glucose metabolism rhythms.<br>  Source: Sophie M T Wehrens et al. "Meal Timing Regulates the Human Circadian System.." <em>Current biology : CB</em>, 2017. PMID: <a href="https://pubmed.ncbi.nlm.nih.gov/28578930/">28578930</a></figcaption>
</figure>

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