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Reactive oxygen species quantification in corneal epithelial cells shows increased oxidative stress following circadian disruption, implicating BMAL1-dependent antioxidant pathways in ocular surface homeostasis.

Figure 12

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Source Paper

Circadian disruption reduces MUC4 expression via the clock molecule BMAL1 during dry eye development.

Experimental & molecular medicine (2024)

PMID: 38956298

DOI: 10.1038/s12276-024-01269-0

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![Figure 12: Reactive oxygen species quantification in corneal epithelial cells shows increased oxidative stress following circadian disruption, implicating BMAL1-dependent antioxidant pathways in ocular surface homeostasis.]()

> Source: Hao Zeng et al. "Circadian disruption reduces MUC4 expression via the clock molecule BMAL1 during." *Experimental & molecular medicine*, 2024. PMID: [38956298](https://pubmed.ncbi.nlm.nih.gov/38956298/)

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  <img src="" alt="Reactive oxygen species quantification in corneal epithelial cells shows increased oxidative stress following circadian disruption, implicating BMAL1-dependent antioxidant pathways in ocular surface homeostasis." />
  <figcaption>Figure 12. Reactive oxygen species quantification in corneal epithelial cells shows increased oxidative stress following circadian disruption, implicating BMAL1-dependent antioxidant pathways in ocular surface homeostasis.<br>  Source: Hao Zeng et al. "Circadian disruption reduces MUC4 expression via the clock molecule BMAL1 during." <em>Experimental & molecular medicine</em>, 2024. PMID: <a href="https://pubmed.ncbi.nlm.nih.gov/38956298/">38956298</a></figcaption>
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