Deskripsi
Melatonin regulates epigenetic modification of CES1 through SIRT1-mediated DNMT1 deacetylation. MLT-mediated CES1 expression induces lipid depletion, leading to ER stress-related apoptosis and blocking intratumoral androgen biosynthesis.
Figure 2
DiagramSource Paper
Melatonin inhibits lipid accumulation to repress prostate cancer progression by mediating the epigenetic modification of CES1.Cite This Figure
![Figure 2: Melatonin regulates epigenetic modification of CES1 through SIRT1-mediated DNMT1 deacetylation. MLT-mediated CES1 expression induces lipid depletion, leading to ER stress-related apoptosis and blocking intratumoral androgen biosynthesis.]() > Source: Lijie Zhou et al. "Melatonin inhibits lipid accumulation to repress prostate cancer progression by ." *Clinical and translational medicine*, 2021. PMID: [34185414](https://pubmed.ncbi.nlm.nih.gov/34185414/)
<figure> <img src="" alt="Melatonin regulates epigenetic modification of CES1 through SIRT1-mediated DNMT1 deacetylation. MLT-mediated CES1 expression induces lipid depletion, leading to ER stress-related apoptosis and blocking intratumoral androgen biosynthesis." /> <figcaption>Figure 2. Melatonin regulates epigenetic modification of CES1 through SIRT1-mediated DNMT1 deacetylation. MLT-mediated CES1 expression induces lipid depletion, leading to ER stress-related apoptosis and blocking intratumoral androgen biosynthesis.<br> Source: Lijie Zhou et al. "Melatonin inhibits lipid accumulation to repress prostate cancer progression by ." <em>Clinical and translational medicine</em>, 2021. PMID: <a href="https://pubmed.ncbi.nlm.nih.gov/34185414/">34185414</a></figcaption> </figure>