Descripción

Structural model of voltage-gated sodium channel (Nav) alpha subunit and phytocannabinoid binding sites. Nav channel blockade is a well-established mechanism of conventional antiepileptic drugs, and some phytocannabinoids show similar activity.

Figure 211

Diagram

Source Paper

Receptors and Channels Possibly Mediating the Effects of Phytocannabinoids on Seizures and Epilepsy.

Pharmaceuticals (Basel, Switzerland) (2020)

PMID: 32751761

DOI: 10.3390/ph13080174

Cite This Figure

![Figure 211: Structural model of voltage-gated sodium channel (Nav) alpha subunit and phytocannabinoid binding sites. Nav channel blockade is a well-established mechanism of conventional antiepileptic drugs, and some phytocannabinoids show similar activity.]()

> Source: Lara Senn et al. "Receptors and Channels Possibly Mediating the Effects of Phytocannabinoids on Se." *Pharmaceuticals (Basel, Switzerland)*, 2020. PMID: [32751761](https://pubmed.ncbi.nlm.nih.gov/32751761/)

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  <img src="" alt="Structural model of voltage-gated sodium channel (Nav) alpha subunit and phytocannabinoid binding sites. Nav channel blockade is a well-established mechanism of conventional antiepileptic drugs, and some phytocannabinoids show similar activity." />
  <figcaption>Figure 211. Structural model of voltage-gated sodium channel (Nav) alpha subunit and phytocannabinoid binding sites. Nav channel blockade is a well-established mechanism of conventional antiepileptic drugs, and some phytocannabinoids show similar activity.<br>  Source: Lara Senn et al. "Receptors and Channels Possibly Mediating the Effects of Phytocannabinoids on Se." <em>Pharmaceuticals (Basel, Switzerland)</em>, 2020. PMID: <a href="https://pubmed.ncbi.nlm.nih.gov/32751761/">32751761</a></figcaption>
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