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Assessment of apoptotic markers in prostate cancer cells following melatonin-induced lipid depletion. ER stress pathway activation is indicated by elevated expression of stress-response proteins.
Figure 11
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Melatonin inhibits lipid accumulation to repress prostate cancer progression by mediating the epigenetic modification of CES1.Cite This Figure
![Figure 11: Assessment of apoptotic markers in prostate cancer cells following melatonin-induced lipid depletion. ER stress pathway activation is indicated by elevated expression of stress-response proteins.]() > Source: Lijie Zhou et al. "Melatonin inhibits lipid accumulation to repress prostate cancer progression by ." *Clinical and translational medicine*, 2021. PMID: [34185414](https://pubmed.ncbi.nlm.nih.gov/34185414/)
<figure> <img src="" alt="Assessment of apoptotic markers in prostate cancer cells following melatonin-induced lipid depletion. ER stress pathway activation is indicated by elevated expression of stress-response proteins." /> <figcaption>Figure 11. Assessment of apoptotic markers in prostate cancer cells following melatonin-induced lipid depletion. ER stress pathway activation is indicated by elevated expression of stress-response proteins.<br> Source: Lijie Zhou et al. "Melatonin inhibits lipid accumulation to repress prostate cancer progression by ." <em>Clinical and translational medicine</em>, 2021. PMID: <a href="https://pubmed.ncbi.nlm.nih.gov/34185414/">34185414</a></figcaption> </figure>