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Receptor occupancy model for phytocannabinoids at opioid receptor subtypes. While direct binding affinity is modest, functional interactions between the two systems appear to be physiologically relevant.
Figure 170
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Receptors and Channels Possibly Mediating the Effects of Phytocannabinoids on Seizures and Epilepsy.Cite This Figure
![Figure 170: Receptor occupancy model for phytocannabinoids at opioid receptor subtypes. While direct binding affinity is modest, functional interactions between the two systems appear to be physiologically relevant.]() > Source: Lara Senn et al. "Receptors and Channels Possibly Mediating the Effects of Phytocannabinoids on Se." *Pharmaceuticals (Basel, Switzerland)*, 2020. PMID: [32751761](https://pubmed.ncbi.nlm.nih.gov/32751761/)
<figure> <img src="" alt="Receptor occupancy model for phytocannabinoids at opioid receptor subtypes. While direct binding affinity is modest, functional interactions between the two systems appear to be physiologically relevant." /> <figcaption>Figure 170. Receptor occupancy model for phytocannabinoids at opioid receptor subtypes. While direct binding affinity is modest, functional interactions between the two systems appear to be physiologically relevant.<br> Source: Lara Senn et al. "Receptors and Channels Possibly Mediating the Effects of Phytocannabinoids on Se." <em>Pharmaceuticals (Basel, Switzerland)</em>, 2020. PMID: <a href="https://pubmed.ncbi.nlm.nih.gov/32751761/">32751761</a></figcaption> </figure>