Figure 211

![Figure 211: Structural model of voltage-gated sodium channel (Nav) alpha subunit and phytocannabinoid binding sites. Nav channel blockade is a well-established mechanism of conventional antiepileptic drugs, and some phytocannabinoids show similar activity.]()

> Source: Lara Senn et al. "Receptors and Channels Possibly Mediating the Effects of Phytocannabinoids on Se." *Pharmaceuticals (Basel, Switzerland)*, 2020. PMID: [32751761](https://pubmed.ncbi.nlm.nih.gov/32751761/)

<figure>
  <img src="" alt="Structural model of voltage-gated sodium channel (Nav) alpha subunit and phytocannabinoid binding sites. Nav channel blockade is a well-established mechanism of conventional antiepileptic drugs, and some phytocannabinoids show similar activity." />
  <figcaption>Figure 211. Structural model of voltage-gated sodium channel (Nav) alpha subunit and phytocannabinoid binding sites. Nav channel blockade is a well-established mechanism of conventional antiepileptic drugs, and some phytocannabinoids show similar activity.<br>  Source: Lara Senn et al. "Receptors and Channels Possibly Mediating the Effects of Phytocannabinoids on Se." <em>Pharmaceuticals (Basel, Switzerland)</em>, 2020. PMID: <a href="https://pubmed.ncbi.nlm.nih.gov/32751761/">32751761</a></figcaption>
</figure>