SleepCited

Restless Leg Syndrome/Willis-Ekbom Disease Pathophysiology.

Richard P Allen
Review Sleep medicine clinics 2015 158 citas

Diseño del estudio

Tipo de estudio
Review
Población
Patients with restless leg syndrome/Willis-Ekbom disease
Intervención
Restless Leg Syndrome/Willis-Ekbom Disease Pathophysiology.
Comparador
None
Resultado primario
Pathophysiology of restless leg syndrome/Willis-Ekbom disease
Dirección del efecto
Neutral
Riesgo de sesgo
Unclear

Resumen

Restless leg syndrome/Willis-Ekbom disease has brain iron deficiency that produces excessive dopamine and known genetic risks, some of which contribute to the brain iron deficiency. Dopamine treatments work temporarily but may eventually produce further postsynaptic down-regulation and worse restless leg syndrome. This article includes sections focused on pathophysiologic findings from each of these areas: genetics, cortical-spinal excitability, and iron and dopamine.

TL;DR

Part of this article includes sections focused on pathophysiologic findings from each of these areas: genetics, cortical-spinal excitability, and iron and dopamine.

Utilizado en revisiones de evidencia